The CTMR was Renewed!
We are thrilled to share that the CTMR was RENEWED! Here’s to another 5 years of supporting muscle research translational muscle research!

We are thrilled to share that the CTMR was RENEWED! Here’s to another 5 years of supporting muscle research translational muscle research!

Congratulations to CTMR faculty investigator Farid Moussavi-Harami, Assistant Professor in the UW Division of Cardiology, for receipt of the Paul F. Cranefield award from the Council of the Society of General Physiologists. The award, reserved for a truly noteworthy paper, is to recognize an independent young investigator who has published an outstanding article in the Journal…
The CTMR is transitioning from X (Twitter) to Bluesky. Follow us on Bluesky Our X (Twitter) account will be closed at the end of January.
This past November, the Center for Translational Muscle Research celebrated another year of pioneering muscle research at the 6th annual CTMR Symposium. This year, the symposium brought together a community of muscle researchers from across the world with a focus on the theme Modeling and Metabolism in Muscle Research.In keeping with the theme, keynote speaker…
We are excited to announce the recipients of the Center for Translational Muscle Research (CTMR) Pilot Award Program for Year 8! This program supports innovative, early-stage research projects that advance our understanding of muscle biology and translate discoveries toward clinical impact. This year’s pilot awards are made possible through the generous support of the National…
Two new grants will accelerate atomic-scale research into the molecular mechanisms of cardiomyopathies. In Autumn 2024, CTMR investigator Matthew Childers was awarded a competitive K99/R00 Pathway to Independence Award from the National Heart, Lung, and Blood Institute of the NIH. Childers’ project combines computational simulations of muscle proteins with stem-cell derived cardiomyocytes to create a…
CTMR Investigators, Mike Regnier and David Mack, were part of a multi-institutional research effort to uncover the disease mechanism for hypertrophic cardiomyopathy caused by a mutation in myosin (MYH7 G256E). The project was a collaborative effort between investigators at UW, Stanford University, University of California Santa Barbara, the Curie Institute in Paris, and the Allen Institute for Cell…