The CTMR was Renewed!
We are thrilled to share that the CTMR was RENEWED! Here’s to another 5 years of supporting muscle research translational muscle research!
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New Grants Power Muscle Science
Two new grants will accelerate atomic-scale research into the molecular mechanisms of cardiomyopathies. In Autumn 2024, CTMR investigator Matthew Childers was awarded a competitive K99/R00 Pathway to Independence Award from the National Heart, Lung, and Blood Institute of the NIH. Childers’ project combines computational simulations of muscle proteins with stem-cell derived cardiomyocytes to create a…
Congratulations to Jeff Chamberlain for receiving the MDA Legacy Award
Congratulations to CTMR faculty member Jeff Chamberlain for receipt of the 2024 Muscular Dystrophy Association (MDA) Legacy Award for Achievement in Research. Jeff is a leading investigator in gene therapy for Duchenne muscular dystrophy (DMD). This award is presented for outstanding contributions to translational neuromuscular disease research and will be presented at the 2024 MDA…
CTMR is Transitioning to Bluesky
The CTMR is transitioning from X (Twitter) to Bluesky. Follow us on Bluesky at: https://bsky.app/profile/ctmr-uw.bsky.social Our X (Twitter) account will be closed at the end of January.
Aditi Prabhala- Husky 100!
Congratulations to Aditi Prabhala for being selected as a 2024 Husky 100 member! The Husky 100 list recognizes undergraduate and graduate students who make the most of their time at UW. Aditi Prabhala is an undergraduate student in the Regnier Heart and Muscle Mechanics (HAMM) Lab mentored by PhD student, Kerry Kao. In addition to her…
New CTMR Program Manager
The CTMR’s Program Manager, Katie Dickinson, is transitioning to support other programs at UW. We will miss Katie in the CTMR. A big thank you to her for skillfully supporting the CTMR over the past year. We wish her all the best in her future endeavors! We are excited to introduce our new Program Manager…
Complex problems require collaborative science
CTMR Investigators, Mike Regnier and David Mack, were part of a multi-institutional research effort to uncover the disease mechanism for hypertrophic cardiomyopathy caused by a mutation in myosin (MYH7 G256E). The project was a collaborative effort between investigators at UW, Stanford University, University of California Santa Barbara, the Curie Institute in Paris, and the Allen Institute for Cell…
