The CTMR was Renewed!
We are thrilled to share that the CTMR was RENEWED! Here’s to another 5 years of supporting muscle research translational muscle research!
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Aditi Prabhala- Husky 100!
Congratulations to Aditi Prabhala for being selected as a 2024 Husky 100 member! The Husky 100 list recognizes undergraduate and graduate students who make the most of their time at UW. Aditi Prabhala is an undergraduate student in the Regnier Heart and Muscle Mechanics (HAMM) Lab mentored by PhD student, Kerry Kao. In addition to her…
Congratulations to Alec Smith!
CTMR Investigator Alec Smith, PhD, recently had an R21 funded through the NIAMS that will be looking at developing human stem cell-based models of the muscle spindle for studying proprioceptive function in skeletal muscle. Additionally, he received an Award from the Weill Neurohub to develop models of the neuromuscular junction for studying amyotrophic lateral sclerosis…
Engineering heart health- featuring CTMR investigators
The remarkable research of CTMR faculty was recently featured on the UW College of Engineering homepage! Read the full story here: Engineering heart health. Congratulations to the Sniadecki, Davis, and Regnier research groups!
Congratulations to Jeff Chamberlain for receiving the MDA Legacy Award
Congratulations to CTMR faculty member Jeff Chamberlain for receipt of the 2024 Muscular Dystrophy Association (MDA) Legacy Award for Achievement in Research. Jeff is a leading investigator in gene therapy for Duchenne muscular dystrophy (DMD). This award is presented for outstanding contributions to translational neuromuscular disease research and will be presented at the 2024 MDA…
CTMR is Transitioning to Bluesky
The CTMR is transitioning from X (Twitter) to Bluesky. Follow us on Bluesky at: https://bsky.app/profile/ctmr-uw.bsky.social Our X (Twitter) account will be closed at the end of January.
Complex problems require collaborative science
CTMR Investigators, Mike Regnier and David Mack, were part of a multi-institutional research effort to uncover the disease mechanism for hypertrophic cardiomyopathy caused by a mutation in myosin (MYH7 G256E). The project was a collaborative effort between investigators at UW, Stanford University, University of California Santa Barbara, the Curie Institute in Paris, and the Allen Institute for Cell…